Hydrocephalus was found after the operation of cerebrovascular hemorrhage, and the operation was cleaned up. I had a fever the next day. The doctor told me to ice it. Is it serious? How long does it take to reduce the fever? (1) 1. First aid principles for acute cerebral hemorrhage ① Prevent further bleeding; ② Reduce intracranial pressure; ③ control brain edema; ④ Specific measures to maintain life function and prevent complications are: (1) Stay in bed quietly: keep the height of the bedside as high as possible and minimize movement; Usually stay in bed for about 3-4 weeks (2). Ensure airway patency: The first five M5 minutes of cerebral hemorrhage are very important to life, because the patient's tongue root falls backwards, which is easy to block the airway. Therefore, it is necessary to ensure airway patency: loosen the collar, take off the denture, lie on your side, so that oral secretions can flow out automatically and clear oral vomit in time; (3) Rational use of sedatives: sedatives and painkillers are used for patients with irritability or epilepsy; (4) Adjust blood pressure: For hypertensive cerebral hemorrhage, use a small amount of reserpine. Consciously give oral antihypertensive drugs (5) and take less exercise: If the patient gets sick in a narrow place, move to a spacious place as soon as possible. The principle is to try not to shake your head and keep your head level to avoid blocking the respiratory tract (6). Medical treatment: the hematoma is small and the intracranial pressure is not significantly increased. Basically, basic drugs are the main treatment, and sometimes drugs to improve cerebral blood circulation can be added at an early stage. Patients with cerebral edema and intracranial hypertension who use traditional Chinese medicine preparations to promote blood circulation and remove blood stasis need active and reasonable dehydration treatment (7). Surgical treatment: most patients with obvious displacement of the large midline structure of hematoma should be operated in time, and sometimes emergency surgery is needed to save critically ill patients. Some scholars believe that hematoma plays a key role in pathological damage, and the volume of ischemic edema caused by hematoma can be several times larger than that of hematoma. Therefore, early operation, even within 6 hours of onset, can minimize secondary damage, improve the success rate of rescue, reduce the disability rate, and thus obtain better curative effect (8). Hemostatic drugs: the dosage of commonly used hemostatic drugs such as phenylethylamine (sensitive to hemostasis), aminobenzoic acid (anti-fibrinolytic aromatic acid) and vitamin K should not be too large. (9) Strengthen nursing and keep the respiratory tract unobstructed: turn over and pat the back regularly to prevent pneumonia and bedsore. Focus on dynamic observation of vital signs, including awake pupil blood pressure and pulse breathing, and measure 1 time every half hour and 1 time every 2-4 hours and carefully record (10). Timely rescue: if the disturbance of consciousness is aggravated or agitated, the pupils are unequal, the light is slow, the pulse is slow, and the blood pressure is high, it means that there is a cerebral hernia and should be rescued immediately. 2. General treatment in acute phase (1) to keep the respiratory tract unobstructed: coma patients can take the head-side position, which is not suitable for supine position, to prevent the tongue from falling back and blocking the airway, turn over and pat the back in time to help expectoration. At the same time, they can often inhale sputum to help the sputum moist. When there are signs of respiratory obstruction, the trachea should be cut in time to avoid hypoxia and aggravate brain edema. Oxygen mixed with 5% carbon dioxide can be inhaled intermittently to avoid inhaling pure oxygen for too long. Pure oxygen can cause cerebral vasospasm and even oxygen poisoning. (2) Maintain the balance of nutrition and water and electrolyte: usually it is better to fast within 1 ~ 2 days after onset, and the daily infusion volume is 1500 ~ 2000 ml, and the intake and output volume are recorded. In addition, we should also pay attention to prevent and correct acidosis, non-ketotic diabetes, hyperosmotic coma or inability to eat. On the third day, you can insert a stomach tube for nasal feeding to ensure nutritional supply, and the daily liquid intake is appropriately limited to 250 ml. 0ml if you have high fever, vomiting, hyperhidrosis and polyuria, you can increase it as appropriate to avoid using high-sugar liquid. If necessary, give fat emulsion injection (fat emulsion), human albumin amino acid or energy mixture (3). Strengthening nursing: Patients with cerebral hemorrhage are in an urgent condition and have a high mortality rate, so nursing in the acute stage is very important. ① Close observation of illness: including consciousness, pupil changes, vomiting, monitoring blood pressure and temperature changes. ② Prevention of complications: The main factor affecting the treatment and prognosis of acute cerebrovascular disease is the prevention of complications. Prevention of urinary tract infection and bedsore is the key point of nursing. 3. Adjust blood pressure. The principle of early antihypertensive treatment for patients with hypertensive cerebral hemorrhage is: (65438+ 0) Grasp the indications of antihypertensive treatment carefully. It is generally believed that when the systolic blood pressure exceeds 24 ~ 26.66 kPa (180 ~ 200 mmHg), appropriate blood pressure reduction can be considered to prevent further bleeding, but patients with high pulse pressure should be careful to reduce blood pressure (2). Blood pressure should be controlled steadily so that the "peak" and "valley" of blood pressure are close within 24 hours, which can not only avoid the damage to blood vessel wall caused by blood pressure fluctuation, but also prevent cerebral perfusion insufficiency that may be caused by hypotension. Antihypertensive therapy should not excessively pursue rapid antihypertensive effect or repeatedly use a large number of powerful antihypertensive drugs, such as reserpine. 0.5 ~ 1mg intramuscular injection of 25% magnesium sulfate 10mg deep intramuscular injection of 6 ~ 12h can be reused, and other oral antihypertensive drugs such as invertase inhibitors or diuretics with strong vasodilation effect should be used with caution or not. When patients are completely insensitive to antihypertensive reaction, we should pay attention to the increase of blood pressure caused by intracranial hypertension (3). The feasible way to reduce blood pressure is to gradually reduce blood pressure to the above level or slightly higher within a period of time without the discomfort of cerebral ischemia. Most people think that blood pressure should be stable at 20 ~ 21.33/12 ~13.33 kpa (150 ~160/90 ~ 65438). It is best to keep the left and right blood pressure slightly higher than the original blood pressure of the patient. (4) Blood pressure circulation and water-electrolyte balance must be closely observed, especially furosemide. At the same time, dehydrated diuretics should be used to reduce intracranial pressure and resist brain edema. By inhibiting sodium in edema tissue from entering cells, brain tissue is indirectly dehydrated and brain edema is alleviated. It has been widely used as a dehydrating agent to treat patients with brain edema, but the situation that continuous high-dose use of the drug causes continuous drop in blood pressure, sharp drop in blood volume and disorder of water and electrolyte must be paid enough attention (5). Pay attention to the changes of blood pressure when using antihypertensive drugs. When the blood pressure is too high, the bedside should be raised by about 30 ~ 45. When the blood pressure is close to normal, we should lower the bedside. If blood pressure continues to be too low, antihypertensive drugs should be used appropriately to maintain the above level. 4. Controlling brain edema and reducing intracranial pressure are important links to prevent the formation of cerebral hernia. After cerebral hemorrhage, the brain edema is gradually aggravated, and it often reaches its peak within 6 hours. After half a month, the brain edema gradually subsided, leading to an increase in intracranial pressure or even. It leads to cerebral hernia, so controlling brain edema and increasing intracranial pressure is the key to reduce mortality. Active measures should be taken in time to control brain edema. When the dehydrating agent is clinically indicated, intravenous injection or intramuscular injection is generally used. Unless the patient is awake, the intracranial hypertension is not serious and there is no vomiting, oral drugs can be chosen. When intravenous injection or oral administration is difficult, rectal drip can also be considered. When severe dehydration and intracranial hypertension occur, 40 ~ 60m mannitol can be injected into carotid artery. L brain tissue can be dehydrated, which has little effect on the whole body. At the same time, the dehydrating agent and its dosage must be considered comprehensively according to the increase of intracranial pressure, heart and kidney function and other general conditions. Strong dehydrating agent should be used in the early signs of deep coma or cerebral hernia. Generally, 2 ~ 3 alternatives should be selected, such as 20% mannitol, furosemide (furosemide) and glycerin preparation. People with heart or renal insufficiency often need to use furosemide first. Colloidal liquids, such as 20% or 25% human albumin, can prevent the decrease of blood volume and avoid hypotension. The application of adrenocortical hormone in acute stage is helpful to alleviate brain edema. Dexamethasone has the strongest anti-brain edema effect, especially on vascular brain edema. The usual dose 10 ~ 15 mg was added to glucose solution or mannitol was given intravenously 1 ~ 2 weeks, and the effect of stopping hormone was slow due to cerebral hemorrhage. Most coma patients are prone to gastrointestinal bleeding and lung infection, and the use of adrenocortical hormone may aggravate or cover up these symptoms. In addition, the hormone can slowly reduce intracranial pressure, and can not quickly resist brain edema. Therefore, routine use is not recommended, especially for patients with diabetes, hypertension and peptic ulcer, which is easy to induce stress gastric bleeding. Gastric mucosal protective drugs should also be used. 5. Application of hemostatic drugs Patients with cerebral hemorrhage have different opinions on whether to use hemostatic drugs. Various hemostatic drugs are mainly used to prevent capillaries in brain parenchyma. The effect of vascular bleeding or oozing blood on arterial rupture bleeding is uncertain. Blind application of hemostatic drugs may increase the risk of myocardial infarction or renal artery thrombosis caused by ischemic cerebrovascular disease in patients with atherosclerosis. Therefore, some people object to the use of hemostatic drugs for patients with gastrointestinal bleeding, but it is necessary to check the coagulation function frequently. Under the supervision of relevant laboratory indexes, patients with cerebral hemorrhage breaking into ventricle or subarachnoid space can be treated with appropriate hemostatic drugs for a short period of time. Anti-rebleeding 6 Artificial hibernation hypothermia therapy can reduce the basal metabolic rate of the brain, reduce oxygen consumption, and improve the tolerance of the brain to hypoxia, thus improving the hypoxia state of the brain, alleviating brain edema, reducing intracranial pressure, protecting brain tissue and reducing or avoiding rebleeding (1). Early hypothermia: try to give medicine for more than 7-8 hours within 6 hours of onset, and the cooling time should not exceed 48 hours. If complicated with high fever, the cooling time can be extended. (2) Cooling method: At present, there are many advanced low-temperature greenhouses. Under limited conditions, it is necessary to adopt the method of head ice cap+arterial ice compress+medicine. (3) The principle of gradual rewarming: stop taking medicine first, then remove the ice compress, and finally remove the ice cap within 8- 12 hours. The complications of this short-term hypothermia are rare, and some of them have muscle fibrillation and irritability, so atracurium can be given. 25 mg or diazepam 10 mg 7. Surgical treatment Because of the wide application of CT in clinic, the diagnosis of hypertensive cerebral hemorrhage has become rapid and accurate. With the development of microsurgery and stereotactic surgery, the surgical accuracy has been improved and the trauma to brain tissue has been greatly reduced. The surgical indications of hypertensive cerebral hemorrhage are constantly expanding. It is generally believed that hematoma will form within 6 hours after the onset of bleeding, and edema will reach its peak after 8-24 hours. It is possible to clear the hematoma before this. Good early surgery for functional recovery can not only clear the hematoma in time to solve intracranial hypertension, but also reduce the damage of blood decomposition to brain tissue, which is of great significance to reduce the mortality and disability rate (1). Surgical indications: There is no uniform standard for the surgical treatment of hypertensive cerebral hemorrhage. It is generally believed that the patient is not particularly old, the function of important organs is good, and there are no serious complications, such as deep coma, gastrointestinal bleeding, decortication, rigidity, mydriasis and central high fever, which meet one of the following conditions: ① The amount of bleeding is more than 20ml; ② Hematoma in thalamus or basal ganglia; ③ If it breaks into the ventricle, puncture and drainage should be performed as soon as possible, and lumbar puncture should be performed at the same time. +0 times /d, release cerebrospinal fluid 10 ~ 20ml each time until the condition is stable. The drainage tube was preserved under strict aseptic operation for about 65438 0 weeks. ④ Hematoma involves the brain stem, and it is not suitable for the elderly or patients with cerebral hernia. ⑤ Preoperative hypertension can be reduced first. ⑤ Use with caution in patients with vascular malformation or ruptured aneurysm. ⑧ Patients with cerebellar hemisphere hemorrhage of about 20ml did not improve after conservative medical treatment. The condition is getting worse or brain hernia appears. (2) Timing of operation: In the past, people thought that the operation of patients with cerebral hemorrhage was critical and the risk of rebleeding was high, and the operation should be carried out after 24 hours. Recent studies show that hypertensive cerebral hemorrhage usually forms hematoma within half an hour of bleeding, but edema around hematoma has not formed for 6-7 hours. The bleeding stopped, edema appeared around the hematoma, and irreversible damage occurred in the brain tissue necrosis around the hematoma, which reached moderate edema within 24 hours and severe edema with the deepening of research. Most scholars advocate early or ultra-early surgery before the occurrence of brain edema around hematoma, that is, surgery within 6-8 hours after the onset, which can not only reduce the pressure of hematoma on brain tissue, but also avoid the occurrence of brain edema, break the vicious circle caused by a series of secondary changes such as blood cell decomposition and brain edema after bleeding, and improve the survival rate and quality of life. It is generally advisable to operate within 3 days after bleeding. Whether puncture is used for bleeding for more than 20 days depends on the specific situation. (3) Surgical methods: The common methods of hematoma removal surgery are: ① neuroendoscopic treatment; ② Minimally invasive surgery for hypertensive cerebral hemorrhage; ③ craniotomy with bone flap or bone window to remove hematoma; ④CT-guided stereotactic aspiration; ⑤ Ventricular drainage hematoma dissolution in 8 cases. Rehabilitation treatment of cerebral hemorrhage The purpose of rehabilitation treatment is to promote the functional recovery of paralyzed limbs and speech disorders, improve brain function, reduce sequelae and prevent recurrence. The time to resume treatment is when the clinical symptoms of brain edema and intracranial hypertension subside and the damaged brain function gradually recovers. In addition to the original drug treatment, the focus should be on improving cerebral blood circulation and promoting nutritional metabolism. The effect of drugs should be eased, and the dose can be gradually increased from low dose to therapeutic dose. Rehabilitation is another important treatment measure in recovery period, especially for patients with severe neurological deficits such as hemiplegia and aphasia, which can achieve better results and significantly reduce disability (1). Prevention of rebleeding: Recurrent cerebral hemorrhage is one of the main causes of death and disability of survivors of cerebrovascular diseases. The interval of recurrent cerebral hemorrhage reported by Song Degen and others in China is 3 months to 5 years, accounting for 65,438+09.5% (58/297) of cerebral hemorrhage in the same period. The recurrence rate was 37.9% within 1 year after the first attack, 75.8% within 2 years and 93% within 3 years, that is, most patients recurred within 3 years. Passeros et al. used joint variable analysis to show that the risk factors of recurrent cerebrovascular disease have no obvious relationship with age, sex, hyperlipidemia, smoking, heavy drinking and diabetes, but the key is uncontrolled hypertension and angioamyloidosis. It is believed that the inducement of rebleeding is hypertension, followed by emotional changes, such as excitement and sadness. Elderly diabetic patients often have a history of TIA or ischemic cerebrovascular disease, and hyperlipidemia rebleeding is rare. Therefore, active control of hypertension, rational treatment of diabetes, attention to self-regulation of emotions, regular diet and timely treatment of constipation are important links to prevent rebleeding. Song Degen and others reported that 58 cases got better after medical treatment and 29 cases died. Cases each accounted for 50% of the patients. (2) Drug therapy: ① Calcium channel antagonist: The nerve cells in the brain tissue around the hematoma after cerebral hemorrhage are in a state of calcium overload. The application of calcium channel antagonists can relieve overload, prevent cell death, improve cerebral microcirculation and increase cerebral blood supply. Commonly used drugs are: nimodipine (Nidal) 20 ~ 40 mg 3 times/d; Or nimodipine (nimotop) 30mg3 times/d; Cinnarizine (Naoyizine) 25mg3 times /d Patients with hypotension, obviously increased intracranial pressure and brain edema should use it with caution ② Brain metabolism activators: drugs that promote nerve metabolism, such as piracetam (Naofukang), cytidine diphosphate choline (CYDP choline), cerebroprotein hydrolysate (cerebrolysin) and ubidecarenone γ-aminobutyric acid (coenzyme Q 10 Diet control: ① nutritious and digestible food should be provided to meet the body's demand for vitamins, inorganic salts and total heat energy in protein; ② Drink plenty of water and often eat semi-liquid food. Because paralyzed patients are often afraid of urinating too much, they should drink as little water as possible, which is not good for patients. They should also have rice and soup in their daily diet, especially porridge. They can also drink more pickles and eat some juicy fresh fruits and fruits properly, which can prevent constipation and urinary tract infection for a few people who don't want to drink water. Occurrence of sexual diseases ③ Pay attention to dietary fiber should not be too fine to prevent constipation. Avoid strong tea, wine, coffee and spicy food ④ Control the intake of salt and cholesterol and increase foods rich in B vitamins (4). Rehabilitation treatment: ① Passive exercise and massage: when the patient's limbs have no muscle strength, passive exercise should be given priority to, and the movements should be light and gentle, and each joint should be performed rhythmically twice a day to maintain the motor function of joints and soft tissues, so as to avoid the limitation of the range of motion due to contracture, and at the same time, the contracture of joints will lead to local blood circulation disorder, which will increase the difficulty of rehabilitation. When the patient's limbs have functions, they will gradually change into a combination of active and passive movements. Special attention should be paid to the abduction and external rotation of the affected shoulder joint during exercise to prevent the pain of shoulder joint contracture ② Active exercise: After the patient's limbs have muscle strength, active exercise and sit-ups should be carried out on the active exercise bed in time. Some patients with cerebrovascular diseases initially regarded themselves as paralysis of the whole limb, not just one side, and felt completely powerless. The first way to overcome this feeling is to help the patient learn to use the healthy limb to move on the bed and stretch and bend the limb in the supine position. When the patient is awake, he should raise the bedside as soon as possible and sit on the bed for physical exercise under the condition of good tolerance, such as pulling the rope, doing sit-ups, reaching out and lifting his legs, etc., so that the tight muscles can be strongly pulled and the range of activities can be increased. Bedside exercise: Patients should gradually learn to sit up beside the bed. The method is that the patient can curl up his healthy side, and then put his healthy lower limb under the affected lower limb, so that the affected limb can be put down from the bedside and sit up with the support of the healthy upper limb. At this time, the patient uses visual afferent nerve and healthy side. It is much easier to learn and train the proprioception of upper limbs after learning to sit in balance. Standing exercise can create conditions in time after bedside exercise, help people turn to stand on their own feet or stand against the wall, and then leave the bed for indoor and outdoor activities. Physical therapy and acupuncture therapy. Medical exercise therapy: physical coordination is caused by frequent and repeated training. When quadriplegia, coordination will be lost, so every time you complete a complex movement, you must be able to perform every simple decomposition movement before coordinating the movement. Only by repeated training from simple to complex step by step and persistently can these muscles become a part of normal activities. For paralyzed muscles, each group of muscles can be improved from simple to complex through active-auxiliary active anti-gravity and resistance exercises. Repeated physical exercise will gradually coordinate the functions of limbs. 9. Stroke unit treatment mode for cerebrovascular diseases (65438+) 0) What is a stroke unit? Stroke unit. It is a management model of stroke treatment, which refers to providing related systematic drug treatment, physical rehabilitation, language training, psychological rehabilitation and health education for stroke patients. The core staff of the stroke unit include clinicians, professional nurses, physiotherapists, professional language trainers and social workers. From the above concepts, we can sum up the characteristics of stroke unit: ① It is not a green channel for hospitalized stroke patients, so it is not the whole process management of stroke, but the hospitalization period of patients. Stroke unit is not a therapy, but a ward management system, which does not contain new treatment methods. The new ward management system should be a multidisciplinary nursing system. That is to say, multidisciplinary cooperation ④ Patients should receive rehabilitation and health education in addition to medication, but the stroke unit is not equal to medication plus rehabilitation, it is a comprehensive eare or organized nursing. The special type ⑤ stroke unit embodies humanistic care and people-oriented for patients. It takes the functional prognosis of patients and the satisfaction of patients and their families as important clinical goals, rather than emphasizing the recovery of neurological function and the improvement of imaging in traditional ward treatment. (2) Stroke unit can be divided into the following four basic types: ① Acute stroke unit. Patients admitted in the acute phase are usually patients within 1 week of onset. In this stroke unit, it is emphasized that the hospital stay of patients should not exceed 1 week. ② Rehabilitation stroke unit. Patients admitted to hospital after 65,438+0 weeks of onset, because of their stable condition, put more emphasis on rehabilitation treatment. ③ Acute and rehabilitation comprehensive stroke unit: also known as comprehensive stroke unit. The common functions of acute phase and rehabilitation phase are combined to treat patients with acute phase, but the hospitalization time from several weeks to several months can be extended if necessary. ④ Mobile stroke unit: also known as mobile stroke group. In this mode, there is no fixed ward patients staying in different wards with multidisciplinary medical teams to make rounds and make medical plans, so there is no fixed nursing team. Some authors believe that this form does not belong to a stroke unit, but only a stroke group [3]. All stroke patients should receive stroke unit treatment: stroke unit is a common stroke medical treatment. It is not difficult to establish a stroke unit. It should be emphasized that all patients must receive treatment in the stroke unit. In order to implement the stroke unit, stroke guidelines in various countries emphasize the inclusion of acute patients in the stroke unit, including the Royal Medical Association (2000) and the European Stroke Association (2000) published in recent years. The guidelines of American Stroke Association (2003) particularly emphasize the necessity of admission (such as early intervention of multi-medical groups in stroke unit rehabilitation). The operation of BOCSS, an organized stroke medical system in Beijing, which was launched in China in 2002, will greatly promote the improvement of stroke medical level in China and its integration with the international community (2). Generally speaking, after 1 week, the hematoma began to dissolve naturally, and the phenomena of brain edema and intracranial pressure increased gradually, and the patient's consciousness gradually decreased. Finally, a few patients recovered well, and most patients left hemiplegia and aphasia to varying degrees. The bad prognosis factor (1) has a large hematoma, and severe brain tissue injury has caused the intracranial pressure to rise continuously (2). Obvious disturbance of consciousness (3) Upper gastrointestinal bleeding (4) Cerebral hernia formation (5) Central high fever (6) Deceptive rigidity (7) Elderly patients over 70 years old (8) Complications of respiratory tract or urinary tract infection (9) Recurrent cerebral hemorrhage (10). With high or low blood pressure and cardiac insufficiency, these patients may be life-threatening or have severe limb paralysis or long-term disturbance of consciousness. 2. Influencing factors of mortality rate of hypertensive atherosclerotic cerebral hemorrhage (1) Generally, the mortality rate of the elderly is high, so we should take a positive and cautious attitude towards the treatment of cerebral hemorrhage in the elderly (2). In the past, there were arteriosclerosis, diabetes, coronary heart disease, emphysema and other important organs, and the reserve function was poor. Poor stress and defense ability are prone to multiple organ failure and high mortality. In the course of treatment and onset, electrolyte imbalance, hypovolemia and iatrogenic factors have seriously affected the normal metabolism of major organs and reduced their functions. (3) Infection is one of the main causes of multiple organ failure and death. Therefore, rational use of antibiotics to control infection is the key to prevent and treat multiple organ failure. (4) Massive upper gastrointestinal bleeding is an important sign of critical illness. The mortality rate of patients with upper gastrointestinal bleeding increased. For people with a history of stomach trouble, especially the hematoma breaking into the ventricle, the latter may be the most dangerous factor leading to upper gastrointestinal bleeding. (5) The death of patients with cerebral hemorrhage is obviously related to the amount of bleeding at the bleeding site and the situation of intraventricular hemorrhage. The greater the amount of bleeding, the more serious the oppression on the surrounding brain tissue, the more obvious the increase of brain edema and intracranial pressure, and the more likely it is to cause the deviation of the midline structure and the compression of the brain stem, leading to the formation of cerebral hernia and death (6). The third and fourth ventricles have more hematocele, which will cause acute obstructive hydrocephalus caused by midbrain aqueduct obstruction and aggravate intracranial hypertension and brain edema. At the same time, bloody cerebrospinal fluid can also directly stimulate hypothalamus, causing neuroendocrine dysfunction, leading to high fever, upper gastrointestinal bleeding, brain-heart syndrome, hyperglycemia and other complications. (7) Hemorrhage of the fourth ventricle can also cause enlargement of the fourth ventricle, which directly oppresses the brain stem, leading to cerebral hernia or respiratory arrest. (8) The mortality rate of patients with hematoma breaking into the ventricle is significantly higher than that of patients without hematoma breaking into the ventricle, and the mortality rate of whole ventricle casting is higher. If the hematoma breaks into the ventricle, the blood clot blocks the cerebrospinal fluid channel, removes the hematoma and continuously drains the ventricle, which can greatly reduce the mortality rate. (9) The direct cause of early death of cerebral hemorrhage is mainly cerebral hernia, so the key to successful treatment is to quickly and effectively relieve brain compression and acute intracranial hypertension. When the occupying effect is aggravated by edema, which leads to the deterioration of nervous system function, active treatment measures should be taken (10). In the treatment of cerebral hemorrhage, it is also very important to reduce the mortality rate. In addition to actively treating the primary disease, comprehensive treatment should be given to supplement daily calories, prevent complications such as upper gastrointestinal bleeding, acute renal failure and secondary infection, and maintain respiratory blood volume, stabilize cardiopulmonary function and regulate blood pressure.