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The clinical manifestations of idiopathic thrombocytopenic purpura tend to thrombosis. What is the principle?
Thrombocytopenic purpura is characterized by significant thrombocytopenia, accompanied by cutaneous mucosal purpura. In severe cases, bleeding may occur in other parts, such as nosebleeds, gingival exudation, menorrhagia or severe hematemesis, hemoptysis, hematochezia and hematuria. Intracranial hemorrhage is the fatal cause of this disease. Henoch-Schonlein purpura is characterized by scattered spots on the skin of limbs or spreading to the whole body. Severe cases are accompanied by joint pain or abdominal pain, bloody stool, vomiting blood, collapse and so on. In severe cases, it can develop into henoch-schonlein purpura nephritis. Thrombocytopenic purpura can be divided into primary and secondary. Primary thrombocytopenic purpura is an immune syndrome and a common hemorrhagic disease. It is characterized by the existence of anti-platelet antibodies in blood circulation, which makes platelets destroy too much and causes purpura; However, the megakaryocytes in bone marrow are normal or increased, which is childish. Clinically, it can be divided into acute and chronic, and its pathogenesis and manifestations are obviously different. First, general treatment: acute and severe patients should be hospitalized. Second, adrenocortical hormone. Adrenal cortical hormone should be the first choice for patients with acute and chronic massive hemorrhage. Splenectomy: Splenectomy is one of the effective treatment methods. Fourth, immunosuppressants. V. Immunoglobulin. 6.danazol. Seven, platelet transfusion. Eight, plasma exchange. Nine, drugs to promote platelet production. Ten, Chinese medicine.