The rapid development of genetic research not only reveals the pathogenesis of many genetic diseases, but also draws up many effective measures to prevent and treat genetic diseases, so that genetic diseases gradually become "treatable diseases", some of which can be controlled by diet conditioning.

Silkworm bean disease is caused by the lack of glucose 6- phosphate dehydrogenase in the body due to genetic factors. Therefore, patients should not eat broad beans and their products, especially fresh broad beans, otherwise it will cause acute hemolytic anemia and be life-threatening in severe cases. It is worth noting that patients with glucose -6- phosphate dehydrogenase deficiency can not only cause hemolytic anemia by eating broad beans, but also be allergic to some drugs, such as primaquine, hexanipine, sulfonamides, furans, antipyretic and analgesic drugs, so be careful when taking drugs. As long as these foods and drugs are avoided, such genetic diseases will not occur. Phenylketonuria is caused by the lack of phenylalanine hydroxylase in the patient's liver. Phenylalanine can not be converted into tyrosine, but only into phenylpyruvic acid, and the concentration of phenylalanine in blood increases. In addition to phenylpyruvic acid discharged from stool, children are called phenylketonuria, which is mainly due to the mental retardation caused by the influence of a large amount of phenylalanine in blood on the development and function of brain cells. To prevent the onset, it is only necessary to take food restriction therapy as soon as possible (within 3 months after birth). After diagnosis, the baby's diet should be mainly rice noodles and milk cakes. As children grow older, they can choose rice, millet, Chinese cabbage, potatoes and spinach. If conditions permit, protein food with low phenylpropionic acid can be specially prepared. Generally around 8 years old, dietary restrictions can be relaxed appropriately.

Galactomia refers to the lack of glucose-1- phosphate uridine transferase in patients, so patients can't use galactose, so they can't feed human milk and milk. Because milk contains lactose, and lactose will produce galactose after decomposition. Excessive galactose level in blood may lead to brain injury, liver cirrhosis, cataract and even death. However, as long as you stop eating dairy products from the day of birth and feed cereal or milk powder substitutes for more than 3 years, you can prevent the disease.

Hepatolenticular degeneration, also known as Wilson's disease, is an autosomal recessive disorder of copper metabolism. It can be divided into "liver type" patients with liver damage as the main symptom and "brain type" patients with nervous system symptoms as the main symptom. Because the disease is caused by copper metabolism disorder, low copper diet is one of the effective measures to treat it. In addition, patients with fructose intolerance need to quit sweets and drinks containing fructose. Patients with hereditary hypoglycemia only need to eat a small amount of sugar several times a day. When people with sickle cell anemia lose water, their cells will become sickle-shaped, so if patients insist on drinking enough water every day, it will help relieve symptoms.