Gastric antral erosive gastritis is a gastrointestinal disease, which is very common in life. Most patients with gastritis attack after a full meal, and there will be symptoms such as stomach pain, flatulence, nausea and vomiting. Patients with erosive gastritis have multiple erosions of gastric mucosa, which may be accompanied by small ulcers. This disease needs to be treated in time, otherwise it will induce other stomach diseases and even endanger life safety. I suggest you recuperate in time. Diet therapy is very important. But diet alone is not enough. To regulate the stomach, we must first remove toxins from the stomach; Then you can recuperate. It is suggested to use tea with tonic flavor, internal conditioning and "stomach" in one step.
1. The seriousness of erosive gastritis is beyond doubt. Failure to treat in time will affect your health. Erosive gastritis can easily lead to bile reflux. If bile reflux occurs, there will be heartburn, acid reflux and other adverse symptoms. As long as the gastric antrum function is seriously damaged, resulting in pyloric closure dysfunction, bile reflux will occur.
2, erosive gastritis can also lead to gastric ulcer, because the gastric mucosa is damaged in a large area, resulting in the loss of the protective layer of the stomach. Gastric acid and bile can always damage the muscle layer of the stomach, leading to serious erosion and development into gastric ulcer.
3, erosive gastritis can lead to gastric perforation, because with the development of the disease, the erosive area is getting bigger and bigger. After the gastric function is seriously damaged, a large amount of gastric acid will be secreted, which will lead to the corrosion of submucosa, and acute gastric bleeding or gastric perforation will be caused after serosa perforation, posing a threat to life and health.
4, leading to atrophic gastritis, erosive gastritis with bile reflux, gastric ulcer, gastric perforation, which can further weaken the self-healing ability of the stomach, reduce submucosal blood flow, reduce submucosal gland atrophy, and form atrophic gastritis.